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Description

Molecular insights into sarcopenia: ferroptosis-related genes as diagnostic and therapeutic targets

NO normally functions to reduce plasma LDL concentration via S-nitrosylation of the LDL receptor adaptor protein ARH, thereby enhancing LDL receptor-mediated clearance of LDL 33

Overlapping genetic factors in NDDs, cancer, and other developmental disorders suggest shared phenotypic genetic heterogeneity as well as common biological pathways and processes (Grove et al., 2019