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However, heterozygotes for CBS defects do not seem to have an increased risk for cardiovascular disease, because occurrence of these mutations is scarce in patients with vascular disease (Kluijtmans et al, 1996)

DNA fragments of SLC7A11 (-1498 to -123) and SLC3A2 (-1407 to -120) promoter regions were amplified from MDA-MB-231 genomic DNA by PCR and subcloned into the pGL3-luc plasmid

Dieses wichtige krpereigene Antioxidans kann freie Radikale ebenfalls unschdlich machen