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Additional antioxidant pathways, including the GTP cyclohydrolase 1 (GCH-1)-tetrahydrobiopterin (BH4) pathway and the ferroptosis suppressor protein 1 (FSP1)-ubiquinone (CoQ)-NADPH pathway, also play vital roles in ferroptosis regulation

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Mechanistic studies showed that GPX4 expression and ferrous iron were increased in 5-FU and AZ628-derived CRC persister cells, residual subcutaneous transplantation tumors, and colorectal cancer patients undergoing neoadjuvant chemoradiotherapy