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There was no increased pancreatitis risk at 6 months, 1 year, 3 years, or 5 years

4.2 Future clinical perspectives Recent work in the field underscores the need for broader and more integrative approaches to AMS therapeutics, recognizing that oxidative stress, inflammation, and disruptions in energy metabolism interact dynamically during exposure to hypobaric hypoxia

While a fibrotic matrix initially contributes to tissue repair, after injury, chronic and persistent kidney damage in the development of CKD leads to uncontrolled fibrotic matrix deposition, resulting in reduced blood supply, destruction of normal organ structure, and eventual glomerulosclerosis, renal tubular atrophy, and interstitial fibrosis [9, 10]

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