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A specific type of TRx (f-type) has been shown to be glutathionylated on a cysteine residue away from its active site, resulting in a decreased activity towards specific target proteins including NADP-glyceraldehyde-3-phosphate dehydrogenase (GAPDH) [189]

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The study revealed that SFN supplementation significantly modulated gene expression profiles in prostate tissue, with upregulation of genes involved in carcinogen detoxification and downregulation of genes associated with cancer progression pathways