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(2015) stated no significant change in UPDRS scores pre- and post-NAC, but the exact scores were not available in the paper

We now demonstrate that GIPR agonist administration critically relies on Gipr expression in the hindbrain, while GIPR antagonism relies on hypothalamic Gipr expressionboth are probably neuronal populations, given that we used hSyn-driven Cre-expression for the region-specific knockout studies

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Moreover, the lower plasma cobalamin levels also were associated with the presence of symptoms consistent with major depressive disorder