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The study concluded that the loss of SCFAs contributes to an imbalance in pro- and anti-inflammatory pathways in the CNS, exacerbating neuroinflammation in CKD (Vaziri et al., 2013)

This mitochondrial dysfunction-induced immunosenescence promotes premature aging phenotypes and multimorbidity, creating a pro-inflammatory environment that simultaneously accelerates atherosclerosis progression and neuroinflammation

The aging metabolome-biomarkers to hub metabolites

The intracellular GSH is decreased with the increased CCF concentration, accompanied by increased tumor cell apoptosis (Figure 5B), which demonstrates the antitumor effect of GSH-promoted CDT