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Further research is needed to determine their impact on clinical treatment outcomes

In innate immunity, Clostridium modulates macrophage polarization (shifting from pro-inflammatory M1 to anti-inflammatory M2), induces tolerogenic dendritic cell phenotypes to suppress excessive antigen presentation, regulates inflammatory body activity and neutrophil hyperactivation, and modulates ILC subsets to reinforce mucosal defense

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In diesem Fall wre eine Glutathion- oder ACC-Substitution allein weniger erfolgversprechend, sondern sie sollte von einer antientzndlichen und/oder antioxidativen Therapie begleitet sein