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Furthermore, semaglutide prevents MASLD/MASH-driven HCC progression in metabolic preclinical models, partly by correcting adipokine imbalances (low adiponectin, high leptin) associated with insulin resistance, which removes a key chronic inflammatory and pro-oncogenic signal linked to the disease ( It is important to note that most of the evidence supporting the anti-tumor efficacy of GLP-1 RAs is derived from preclinical studies, including in vitro studies and animal experiments demonstrating reduced proliferation of the cancer cells, enhanced apoptosis, and modulation of oncogenic signaling pathways ( These systemic and direct molecular effects, particularly the potent anti-inflammatory actions and metabolic correction, are highly relevant to mitigating the progression of both ALD and MetALD
[DOI] [PMC free article] [PubMed] [Google Scholar] 139.Pangalangan J, Puma J, Tollefson M, Frates B
These could be hospital records indicating visits due to persistent nausea, abdominal bloating among others which are typical signs of gastroparesis
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