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It binds to these toxins and facilitates their transport through bile or urine for excretion

These systems metabolize and scavenge for reactive oxygen species, in a controlled and precise fashion

Conclusion PLB ameliorates DHT-induced mitochondrial dysfunction and ferroptosis in KGN cells by inhibiting YTHDF1 expression, and its action is dependent on the mechanism by which YTHDF1 regulates SLC7A5 translation through m6A modification

To achieve cell-type specific knockdown, we prepared a mixture of adeno-associated virus (AAV)-GFAP104-Cre-mCherry virus with either Lenti-pSico-scrambled-EGFP (control) or Lenti-pSico-MAOB - shRNA-EGFP (MAOB KD)