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As a Th2 effector, IL-31 correlates with itch severity and signals via IL-31 receptor alpha (IL-31RA)/Oncostatin M receptor beta (OSMR) receptors on sensory nerves to release calcitonin gene-related peptide and drive neurogenic inflammation (Lee et al., 2018)

This process is driven by the inactivation of glutathione peroxidase 4 (GPX4) and the pathological accumulation of lipid hydroperoxides (LPOs) within cellular membranes (Stockwell et al., 2017

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During colorectal cancer progression, inflammation-induced mitochondrial ROS drive malignant transformation through dual mechanisms