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Then, independent of allowing you neurons to get this easier source of energy...if the neurons are using glucose because they need energy but the glucose can't be used to repair that damage to the pentose phosphate pathway, I think that's one component of it in terms of the temporal effects, like how soon after the damage

TAMs in this subgroup drive T cell exhaustion by secreting SPP1, thereby weakening anti-tumor immunity

(A) qPCR data showing the normalized mRNA expression of NRF2-regulated genes ( Gclm , Nqo1 , Srxn1 , Hmox1 , Gsr ) in RAW264.7 Ms with (+) and without (-) oxLDL loading and LPS stimulation for 6 h (n = 45)