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Alteration of gut microbiota in individuals at high-risk for rheumatoid arthritis associated with disturbed metabolome and the initiation of arthritis through the triggering of mucosal immunity imbalance

Primary Cellular Pathways Folate-AICAR-AMPK Pathway Activation Research has demonstrated that MOTS-c directly inhibits the folate cycle and its tethered de novo purine biosynthesis, leading to accumulation of 5-aminoimidazole-4-carboxamide ribonucleotide (AICAR), a well-characterized AMPK activator

Key mechanistic observations from laboratory studies: G-actin sequestration: TB-500 binds monomeric G-actin, regulating the pool available for filament polymerization

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