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doi: 10.1016/j.redox.2017.09.009

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Following pioneer observations that the E2F1-CDK4-pRb pathway is regulated by glucose and insulin in pancreatic islets (17), we show here that the treatment of pancreatic islets with a Glp1r agonist modulates E2F1 pathway through the phosphorylation of pRb and the activation of the E2F1 transcriptional activity