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This supports our findings where MTX injection evoked oxidative stress with augmented hepatic lipid peroxidation product while declining hepatic antioxidant capacity as depicted by lower GSH and SOD

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For instance, it remains undefined whether Gd-IgA1 directly binds to mesangial cell receptors to activate the PPARFABP1 axis, or if indirect signals (e.g., inflammatory cytokines and complement activationhallmarks of IgA nephropathy) are required