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By mimicking the natural incretin hormone GLP-1, these medications activate specific receptors throughout the body, triggering glucose-dependent insulin secretion, suppressing glucagon release, slowing gastric emptying, and modulating appetite centres in the brain

The knowledge that ESR2 receptors expressed in trigeminal ganglion and regulates estrogen effects on neuroinflammation can explain its influence on pain chronification

Their use is associated with better glycemic control and a low risk of hypoglycaemic events in patients for whom other oral antidiabetic drugs (OADs), such as metformin, are not sufficient to achieve adequate glycemic control [1]