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To explore the relevance of GSH to the distinct redox properties of FoB and MZB in vivo, we generated B cell-specific Gclc -deficient mice by crossing Gclc fl/fl mice with Mb1-Cre + mice ( Gclc fl/fl Mb1-Cre + ), in which the Cre recombinase gene is expressed under the control of the B cell-specific promoter Mb1 46

MEK, in turn, phosphorylates and activates mitogen-activated extracellular-signal-regulated kinase (ERK)

Here, we employed two different experimental settings in which glutathione and NADP redox responses are diametrically opposed to test whether NERNST responds predominantly to NADP or glutathione redox changes

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