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Pathogenic loss-of-function variants in SLC52A2 or SLC52A3 abrogate membrane uptake of riboflavin, precipitating secondary FMN/FAD depletion in metabolically demanding neurons and glia (13, 14)

These references provide a comprehensive overview of semaglutide use in managing type 2 diabetes mellitus, with a focus on potential associations with Hashimotos thyroiditis, aiding healthcare providers in assessing its safety profile in patients with autoimmune thyroid disorders