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Our results provide a possible mechanism in which upregulation of phosphorylated p53 and p16 maintain the cell cycle arrest of keloid fibroblasts, and FOXO4-DRI selectively induces apoptosis of keloid senescent fibroblasts by prompting p53-pS15 nuclear exclusion

Taken together, our data suggest hepatic GCLC gene expression is increased during rapid growth and since an increase in GCLC expression alone can lead to increased GSH level, hepatocytes may express more GCLM than GCLC

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