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Although the exact etiology remains unknown, current evidence points to a significant genetic component in the development and severity of FM which can be associated with a spectrum of diseases, including infections, diabetes, rheumatic diseases, and psychiatric or neurological comorbidities [6]

When we stop questioning, we are tempted by simplistic explanations: miracle cures, absolute claims, or declarations that ignore biological limits and trade-offs

Animal studies demonstrate that BPC-157 administration following induced injury results in measurably lower inflammatory marker levels compared to untreated control groups

PAK2 heterozygous mice exhibited reduced spine density and impaired LTP (Wang et al., 2018)