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The senolytic demonstrates preferential elimination of transforming growth factor- (TGF-)-induced myofibroblasts while sparing normal fibroblasts, showing selectivity for pathological senescent cells driving fibrosis

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The combination addresses different but complementary repair pathways: BPC-157 promotes growth factor signaling and angiogenesis through the VEGF pathway, while TB-500 facilitates cellular migration and differentiation through actin modulation