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The majority of circulating T3 is generated not directly by the thyroid, but via peripheral deiodination of T4 by iodothyronine deiodinasesprimarily DIO1 in the liver and kidneys, and DIO2 in the brain, pituitary, and skeletal muscle [16,17]

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Without explicit distinction between these levels, grouping such outcomes together under the broad label of taste change may generate interpretive confusion