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Similarly, endothelium-mediated vasodilator responses were restored when a transgene for GPx1 was introduced into the CBS +/ mice by crossbreeding transgenic GPx1 overexpressing mice and CBS deficient mice, suggesting that increased expression of GPx1 could compensate for the negative effects of homocysteine on bioactive NO [60]
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doi: 10.1074/jbc.M310171200
Consistently, GSH application on leaf 1 significantly induced the expression of defense-responsive genes ( OPR3 , JAZ5 , and JAZ10 ) in leaf 6 (by ~5.6, ~8.6 and ~5.7 folds), which is comparable to that induced by Glu (Fig
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