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303 From the observations in this study it seems that GLP-1R knockout mice adapt to the absence of GLP-1R, and are able to maintain glucose tolerance and desirable plasma insulin levels in response to glucose challenges

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(2024) demonstrated that Emodin confers protection against severe AP-associated lung injury by activating the Nrf2/HO-1/GPX4 signaling pathway and inhibiting ferroptosis, underscoring the pivotal role of oxidative stress in mediating not only pancreatic injury but also dysfunction in distant organs