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Our literature review has shown studies reporting tau deposits in tissue samples of the brain from autopsies of COVID-19 patients compared to the control group, and the in-vitro human brain organoid model has shown aberrant phosphorylation of tau protein in response to SARS-CoV-2 infection

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In the myocardial injury model, cardiac fibroblast-derived Exos inhibit SLC7A11 expression and stimulate cardiac ferroptosis by delivering miR-23a-3p [223]