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SVZ-derived progenitors have a strong migratory potential and are equipped to respond to inflammatory cues, but their contribution to myelin repair is unlikely to extend very far from periventricular structures

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For example, mutant SOD1 produces abnormal SOD molecules which are theorized to serve as the seed for large clusters of misfolded proteins that are toxic to neurons (Karch 2009

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10.1111/j.1471-4159.1980.tb11272.x J

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