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This article identifies the sources and targets of GLP-1 in the brain and review the mechanisms which mediate its neuroprotective effects, as well as implications for Alzheimers disease (AD) and Parkinsons disease (PD)

In contrast, intracellular NOD-like receptors (NLRs), such as NOD1/NOD2, primarily sense bacterial peptidoglycan fragments (e.g., iE-DAP and MDP)

La activacin simultnea de ambas vas permite que el agonista dual de los receptores GLP-1/GIP ejerza un efecto sinrgico, ofreciendo ventajas potenciales en la reduccin de la hemoglobina glucosilada (HbA1c), el control del peso corporal y la optimizacin de diversos parmetros metablicos

thus, decreased levels can make them more susceptible to damage from oxidative stress, leading to rupture and causing hyperbilirubinemia and anemia (6)