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) increases fasting GLP-1 secretion 1.8-fold compared to saline control, reaching 6 pmol/L at 120 min, which increases even further in leptin-deficient mice ( ob/ob ) (Sivitz et al., 1997), which may link the late-phase GLP-1 secretion induced by both leptin and water

Tumor-like stem cells derived from human keloid are governed by the inflammatory niche driven by IL-17/IL-6 axis

As the results, interestingly, GLP-1 receptor expression was not decreased at all after acute exposure to increased glucose and/or cholesterol levels (Supplemental Fig

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