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During dialysis, patients experience subnormal plasma/serum free carnitine concentrations [13] with plasma levels dropping by 60% with a slow replacement from carnitine stores such as from skeletal muscle during the interdialytic period

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Similarly, GSH-activatable synthetic ion channels undergo GSH-mediated disassembly to release channel-forming compounds, leading to caspase-dependent apoptosis 217

It is clear now that each increase of 5mol/L in homocysteine level increases the risk of coronary heart disease events by approximately 20%, independently of traditional coronary heart disease risk factors