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This structure provides the anatomical basis for the realization of bidirectional communication and functional interaction between neurons and SGCs, thereby enabling SGC-mediated regulation of synaptic plasticity ( Studies have shown that purified IgG extracted from FMS patients with FMS specifically binds to rat dorsal root ganglia (DRG), with nine distinct binding clusters identified, suggesting that autoimmune antibodies may participate in FMS mediated by SCGs ( + SGCs to induce NPP ( 7 Central glial cells contribute to NPP development Concomitant activation of microglia and astrocytes observed in a reserpine-induced FMS rat model underscores the pivotal role of central glial cell activation in NPP ( In 2022, a study investigating cortical morphological remodeling in patients with CPLBP and FMS revealed that NPP may be associated with structural remodeling of the myelinated architecture in the limbic system cortex

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