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A cross-sectional study of older adults treated with semaglutide reported a prevalence of reduced muscle mass of 27.7%, reflecting the high baseline risk in this population rather than drug-specific effects [24]

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Similarly, oxidation of the extracellular thiol redox potential stimulates proliferation and pro-fibrotic signaling of transforming growth factor beta-1 (TGF1) within fibroblasts (269) and also activates NF-B and stimulates monocyte adhesion to vascular endothelial cells (111)