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Senolytic therapy alleviates A-associated oligodendrocyte progenitor cell senescence and cognitive deficits in an Alzheimers disease model
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Buffoni F, Pino R, Dal Pozzo A
3.2 Modulation of key molecules The metabolic reprogramming of TECs during AKI is regulated by an intricate signaling network involving AMP-activated protein kinase (AMPK), mechanistic target of rapamycin complex 1 (mTORC1), sirtuins (SIRTs), PPAR, PGC-1, and hypoxia-inducible factor-1 alpha (HIF-1)
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