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The incorporation of -methyl leucine at position 13 serves as a key structural element that enhances both glucose-dependent insulinotropic polypeptide and glucagon receptor activation while maintaining glucagon-like peptide-1 receptor affinity [7] [9]

Hruby received a number of academic awards for work in this area

Mitophagy is impaired in HF, causing the accumulation of damaged mitochondria, due in part to reduced PGC-1 expression, and overexpression of PGC-1 restores mitochondrial function and cardiac performance in HF models [18]