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19 (Elsevier, 1995) Fredriksson, R., Lagerstrm, M

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Nov 5 2019;25(6):673-693

While the phenotypic insulin resistance in NAFLD would be expected to counteract insulin-mediated SREBP1c activation, a state of selective insulin resistance ensures that insulin retains its ability to promote DNL through SREBP1c while being unable to suppress gluconeogenesis [41]