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-cell-selective knockout of the GLP-1R in mice with impaired glucose tolerance after intraperitoneal injection of glucose and exogenous supplementation of GLP-1 did not induce insulin secretion, but glucose tolerance returned to normal after oral glucose, suggesting that the extra-islet GLP-1R plays a regulatory role in oral glucose tolerance (Smith et al., 2014)
Nature 579 , 586591 (2020)
The overlapping constipation risk deserves particular attention because severe constipation is one of the most common reasons people discontinue GLP-1 receptor agonists
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