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Mechanistically, L-tryptophan upregulated the expression of GLP-1related genes (e.g., Gcg and Pcsk1 ) in intestinal L cells, further promoting GLP-1 production ( In contrast, some postbiotics may suppress GLP-1 signaling

Published by Cambridge University Press on behalf of The Nutrition Society Non-alcoholic fatty liver disease (NAFLD) has emerged as the most prevalent hepatic disorder worldwide and is estimated to afflict approximately 38 % of the worlds population, with an annual incidence of 4613 new cases per 1000 person-years (Reference Wai-Sun Wong, Ekstedt and Lai-Hung Wong1,Reference Le, Le and Baez2)

This was associated with a significant reduction in the glucose excursion by 30% (95% CI -34.53, -25.45) in the high GLP-1 and 21% (95% CI -25.62, -16.55) in the low GLP-1 group, giving rise to a significant between-group difference (p = 0.01) ( Figure 2F )

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