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Furthermore, it also induces the expression of mitochondrial transcription factor A (TFAM), a controller in replication and transcription of mitochondrial DNA, to further drive mitochondrial biogenesis (Jornayvaz and Shulman, 2010

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These results indicate that the infarcts generated from the ischemia/reperfusion procedure are platelet dependent in nature and support the importance of GPx-3 deficiency in modulating this platelet-dependent response

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