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In clinical studies, GSH and GSSG are most often measured in whole blood or in isolated red blood cells (RBCs) based on the assumption that, although indirect, this minimally invasive type of analysis provides valuable information on the redox balance of less accessible tissue and organs and of the whole organism [7]

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Elevated ROS levels and activation of the oxidative damage-associated p38 MAPK signaling pathway were also observed in the LCs of premature aging mouse models, with partial restoration of steroidogenic capacity upon treatment with p38 MAPK inhibitors [39]