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This observation led to the development of (Pro3)GIP, an analog of GIP with a glutamic-acid-to-proline substitution at position 3, improving glucose homeostasis and preventing weight gain in animal models of diabetes and obesity (Sparre-Ulrich et al., 2016)

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In the -cell, PC1/3 is up-regulated, resulting in increased processing of proglucagon to GLP-1 while DPP-4 is down-regulated in order to reduce degradation and enhance local intact GLP-1 concentrations