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It is thought that portal hypertension (high blood pressure in the livers venous system) due to NAFLD may impair the kidneys ability to regulate sodium levels and blood pressure, triggering dysfunctional compensatory mechanisms and setting the stage for CKD

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Additionally, in the acute lung injury model, significantly upregulating the expression of FXR can inhibit the phosphorylation of p38 MAPK and NF-B p65, reduce the release of inflammatory factors, and thereby protect lung tissue (108)