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In both types of cells, upon ER stress induction by thapsigargin (Tg) treatment, all the three UPR pathways were activated, as determined by phosphorylation of IRE1, PERK, eIF2, and cleavage of ATF6 to generate the activated form, as well as increased expression of the downstream transcription factors XBP1s and ATF4 (Fig

The observed waning of endogenous incretin enhancement over time following LSG, particularly in patients experiencing WR, suggests that individual variations may influence the durability of surgical outcomes in gut hormone adaptation