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The jury is out at the moment, but their potential to be differentiated from injectable peptides is clear and seems likely to drive significant research into small molecule modulators of GLP-1 for the treatment of diabetes, obesity and perhaps addictive behaviours for many years to come.

Human studies of glucagon receptor antagonism show a striking result: hepatic amino-acid clearance drops hyperaminoacidaemia develops skeletal muscle becomes the fallback source of amino acids This pattern mirrors the very changes we see under GLP-1RA therapy

Is the issue obesity or insulin resistance

Medication interactions should also be considered