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The exenatide-treated group mainly highlighted a predominant enrichment of NF-B signaling, suggesting that exenatide may exert its effects by modulating key regulators of inflammation [64] (Fig

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207 Liraglutide treatment was also demonstrated to inhibit beta-cell apoptosis in isolated human pancreatic beta-cells, and after 24 hours, the islet beta-cell proliferation rate tripled