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Overall, whilst these findings imply that increased production of ROS is associated with C9orf72-ALS, it is unclear whether loss of C9orf72 or expression of hexanucleotide repeat expansions disturbs redox homeostasis

doi: 10.1210/jc.85.11.4053 48 GalsgaardKDWinther-SorensenMOrskovCKissowHPoulsenSSVilstrupHet al

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Secondary bile acids are also potent stimuli for PYY secretion and the mechanisms by which this occurs are consistent with those for GLP-1 secretion (Kuhre et al., 2018)