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Together, these findings indicate that reactive nitrogen species promote TTR crosslinking through reversible thiol activation, whereas strong oxidants divert cysteine chemistry toward stable overoxidized states that suppress disulfide-mediated conversion

PMID: 253 MehmoodANawabSJinYHassanHKaushikACWeiDQ

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Figure 2 Atherosclerosis, a chronic inflammatory disease, may be an ideal environment for the high viral replication capabilities of SARS-CoV-2 in human cells, enhancing hyper-inflammation secondary to immune system dysregulation (Figure 3) that leads to adverse outcomes, as shown in patients with cardiovascular risk factors