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Manipulating CSE and ETHE1 levels in both directions, we found a consistent pattern in that the two enzymes mirrored each other: silencing CSE yielded the same effect (slower radical removal) as overexpressing ETHE1, and overexpressing CSE yielded the same effect (faster radical removal) as silencing ETHE1 (Fig
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KPV exerts its anti-inflammatory effects through intracellular pathways, independent of melanocortin receptors (MC-Rs)
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