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Unlike GLP-1 agonists that reduce appetite or growth hormone peptides that stimulate lipolysis, the compound addresses fat metabolism at the enzymatic level: it removes the molecular governor that locks adipocytes into a low-energy-expenditure state, potentially restoring normal metabolic function in fat tissue where NNMT is overexpressed

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That would allow us to give each one what they need, or to propose a sequential treatment, first the triple agonist, first the triple agonist, then tapering to double, and finally to a single hormone. Manuel Tena, professor of Physiology at the University of Crdoba in Spain, notes that with these drugs, too much emphasis is placed on weight loss, but the effects go far beyond that, from improving the metabolic profile to enhancing insulin resistance or even reversing type 2 diabetes. However, he cautions that there is a therapeutic consensus that the application of these drugs must be paired with lifestyle changes, and the therapeutic arsenal must be reserved for cases where obesity doesnt respond to other, more common measures, such as dietary improvements or increased physical activity

The goal is to help patients remain on an effective therapy long enough to achieve sustained metabolic and cardiovascular benefit without unnecessary interruption