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This was followed by a 35-day pharmacokinetic blood sampling and safety assessment period (i.e., 45 times the estimated cagrilintide t 1/2 ), with blood samples collected on days 8, 15, 22, 29 and 36

Lp(a) levels are estimated to be 90% hereditary .[ref] (Thats really high when it comes to hereditary estimates!) High Lp(a) increases the risk of cardiovascular events: Across large cohorts, higher Lp(a) roughly doubles the risk of heart attack, aortic stenosis, or major cardiovascular events compared to low Lp(a)

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