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In pancreatic KRAS-mutated cancer cells, which can proliferate using albumin as the extracellular source of leucine, loss of SLC38A9 or its transport function strongly inhibited mTORC1 activation by macropinocytosed albumin as evidenced by cell proliferation and tumor formation (Goberdhan et al., 2017)

Oxidative stress has been suggested to play a key role in carcinogenesis

This approach holds promise for the design of personalized treatment regimens, offering significant potential in addressing complex metabolic disorders and cancer