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In spite of these copper-rich senile plaques found in AD (Bagheri et al., 2018), we see evidence of a more generalized brain-wide copper deficiency that is central to this disease (Xu et al., 2017), that has been postulated to be due to the copper binding ability of A leading to neuronal copper deficiency, a process that is exacerbated by the increased copper requirement in microglia (Bagheri et al., 2022)

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En fibroblastos de tendn se describe estmulo de migracin y outgrowth celular va la ruta de sealizacin FAK-paxillin, con formacin de F-actina y mayor supervivencia bajo estrs oxidativo

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